By Eberhard Nieschlag, Hermann M. Behre, Susan Nieschlag

This is often the third, absolutely revised version of a well known textbook that maintains to symbolize the most useful within the literature on medical andrology. It examines intensive all elements of male reproductive healthiness, encompassing the elemental body structure of male reproductive functionality and quite a lot of problems. all the chapters is written by means of striking specialists within the box. one of the subject matters lined are: Testicular functionality, sperm maturation and fertilization, diagnostic techniques, infertility, fundamental and secondary hypogonadism, late-onset hypogonadism, erectile disorder, genetic issues, environmental affects, psychology and sexual drugs, testosterone treatment, assisted copy: TESE and ICSI, cryopreservation, male birth control and ethics in andrology.

This quantity will function a textbook for the newcomer to andrology, whereas delivering the skilled health professional with a worthwhile reference paintings. it is going to attract all who're interested in male reproductive health.

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Additional resources for Andrology: Male Reproductive Health and Dysfunction

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The various germ cells are arranged in typical cellular associations within the seminiferous tubules known as spermatogenic stages (Fig. 5) and the entire spermatogenic process can be divided into four phases: 1. Mitotic proliferation and differentiation of diploid germ cells (spermatogonia) (spermatogoniogenesis) 2. Meiotic division of tetraploid germ cells (spermatocytes) resulting in haploid germ cells (spermatids) 3. Transformation of spermatids into testicular sperm (spermiogenesis) 4. Release of sperm from the germinal epithelium into the tubular lumen (spermiation).

The complete duration of the human spermatogenic process is still not entirely clear (Amann 2008). Ad = A-dark spermatogonium (testicular stem cells, divides rarely), Ap = A-pale spermatogonium (self-renewing and progenitor cell for spermatogenesis), B = B spermatogonium, Pl = preleptotene spermatocytes, L = leptotene spermatocytes, EP = early pachytene spermatocytes, MP = mid pachytene spermatocytes, LP = late pachytene spermatocytes, II = 2nd meiotic division, RB = residual body, Sa1–Sd2 = developmental stages of spermatid maturation G.

Testosterone is already produced by the fetal testicle during the 10th week of gestation, under the stimulation of fetal LH and maternal hCG. 30 The role of maternal hCG in this crucial phase of gonadal development is suggested by the fact that a mutation of the LH ß chain leading to a biologically inactive gonadotropin is associated with normal sexual differentiation (Huhtaniemi et al. 1999). Conversely, inactivating mutations of the LH receptor produce a clinical syndrome resembling complete androgen insensitivity, with a phenotype of female external genitalia (Themmen et al.

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